What is Alphabolin and what does it do?

Alphabolin – also known as Tapostolin – is an oral steroid. The chemical formula is C 6 H 10 NO 2. The drug itself is derived from the plant Equisetum acidum. Primobolan – also known as Metabolist – is an injectable form of the hormone Enantiol which works in a very different way from Alphabolin. Alphabolin and Primobolan are chemically similar but there are some important differences.


Alphabolin uses in Bodybuilding

The main function of primobolan depot is to stimulate testosterone production in men. In this respect it resembles Deca-Hydrocortisone (cortisol), which is also a corticoid. The major difference however lies in the fact that it does not have any side effects. Its mechanism of action is similar to that of Enantiol. It stimulates the production of testosterone, the male hormone, in the body by binding to the receptors on the pituitary gland.

Like Enanthol, alphabolin binds with the receptor for testosterone propionate and inhibits the action of estrogen. Thus, when the pituitary gland is stimulated, the production of estrogen is reduced. Primobolan bayer is useful for treatment of both conditions (the reduction of estrogen and the inhibition of testosterone) and has recently been introduced as a medication in generic form. The generic name of the drug is methenolone propionate.

Bands of amino acids are arranged in a headspace or ring in order to generate a sequence of letters called a peptide. The modern technology has greatly developed the manufacturing process of peptides and several of them are now used in order legal anabolic steroid products like boldebolin alpha. This particular compound has not yet been patented but its manufacturer, GenPharma, has filed for a US patent. A lot of research has been conducted to identify if the use of this novel compound is safe and efficient.

Research of Aphabolin

The first research was conducted on human volunteers who had undergone liver or kidney injuries. Cypionate or alphabolin (both synonyms for this chemical compound) was administered intravenously. It was found that patients who were administered alphabolin showed no significant changes in their body weight or blood chemistry values except for a slight increase in plasma antioxidant levels.

This result is quite contrary to the claims of some manufacturers that anavar 10 mg (50 tabs) will effectively reduce lactic acid production in the body. Therefore, further clinical trials on larger groups of subjects are necessary to establish whether this compound has any useful medical use.

Based on the results of the present study, a phase 2 clinical trial is currently under way in the United Kingdom. Analgesia caused by local administration of alphabolin acetate was compared with the effect of intravenous lactic acid and found to be significantly more efficient. Another comparative study between analgesia caused by alphabolin acetate inversed and sham is underway at another hospital. It is believed that micro and primoblan oral ulcers represent distinct and separate entities. Therefore, the exact mechanism of action between these agents is not clearly known.

Analgesics or NSAIDs that act on the non-neovascular sites of the body have been widely used in the treatment of inflammatory and autoimmune diseases. It has been found that combining alphabolin with methenolone may improve the efficacy of such treatments. Studies have shown that oversedation of the administration of alphabolin into the ivary can reduce pain and the associated neuralgia (nerve damage) in patients who have undergone spinal surgery.

When administered into the spinal canal, it increased the permeability of the cervical fluid and allowed increased diffusion of pain signals. Similarly, inversed administration of alphabolin into the anteroderm, which produces no pain, reduced pain and swelling after a spinal injury in patients who had undergone chiropractic treatment. It is, however, essential to note that these results were obtained in patients who were under post-operative general anesthesia, and therefore were in a state of severe pain.


The general conclusion from the present study is that although the administration of alphabolin and methenolone does not result in significant differences in the efficacy of therapy for acute or chronic pain, they have distinct pain reducing properties when given separately. When administered into the spinal cord, both substances cause similar changes in the spinal cord as reflected in the data collected during tests.

However, the mechanism of action for alphabolin and methenolone is different and it is for this reason that the effectiveness of one drug is more reliable than the other when given alone. It was found that the combination of alphabolin and methenolone was more effective than the administration of either substance alone. This suggests that the nervous system acts synergistically with the metabolism in order to produce the changes seen in the spinal cord. However, further studies are required to clarify whether these findings are applicable to other pain states.

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